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Furthermore, a correlation between physical disability and performance in some BRB-N tests was observed.

BrB-cine images generally show larger lumen areas than BB-cine images.

Conclusion: COP and BRB can be absorbed across intestinal epithelial cells, and they are completely absorbed compounds.

Purpose: Inflammation associated with blood-retinal barrier (BRB) breakdown is a common feature of several retinal diseases.

Breakdown of the endothelial junctional complexes in the BRB negatively affects neuronal signaling and ultimately causes vision loss.

The BRB diet impacted colon mucosal microbial composition with a more robust effect observed on the luminal microflora.

The developed retinal microvasculature-on-a-chip will enable improved understanding of BRB biology and provide an additional tool for drug discovery.

Diabetes affects the physiology and function of retinal vascular cells including astrocytes (AC) leading to breakdown of BRB.

Results: Exendin-4 dramatically reduced the BRB permeability induced by IR injury, which was associated with suppression of inflammatory gene expression.

EC-proNGF expression induced vascular permeability including breakdown of BRB and albuminuria in the kidney without affecting VEGF level at 4-weeks.

Thus, we suggest that RAGE could be a potential therapeutic target for intracellular Aβ induced outer BRB breakdown in AMD.

Identifying the factors that contribute to BRB formation during human eye development and maintenance could provide insights into such diseases.

Breakdown of the blood-retinal barrier (BRB) occurs in several retinal diseases and is a major cause of visual loss.

Compared with the retina during BRB maturation, AKAP12 expression in retinoblastoma patient tissue was markedly reduced whereas that of VEGF was increased.

The Symbol Digit Modality Test (SDMT) best correlated with the other individual tests and contributed most to the general BRB-N factor.